Pathophysiology of Acute Kidney Injury
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Pathophysiology of Acute Kidney Injury
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Atkin and coauthors use a uniquely rich dataset from Mexico, and find that the arrival of global retail chains led to reductions in the incomes of traditional retail sector workers, but had little impact on average municipality-level incomes or employment; and led to lower costs of living for both rich and poor households.
Although it has been reported that a targeted mutation of the c-met receptor in collecting duct impairs recovery from transient ureteral obstruction and hastens the development of fibrosis.
Migration and extracellular matrix Severe renal injury leaves behind denuded areas of the basement membrane and also activates a variety of proteases with the potential to degrade the tubular basesment membrane.
Nuclease activation represents a point of no return in the injury process.
This will help you see that, over the long run, growth has roughly followed an exponential path.
Adaptive nature of kidney and proximal tubules As described above the cellular response to injury is heterogeneous, with some cells undergoing necrosis or apoptosis, while others are sublethally injured.